There has been an increase in the balance between the inflammatory and reparative leucocytes. These differences only play a vital role in allowing for the optimal healing process and hence, play a vital role in myocardial infarction. There has been an increase in the quantity of heterogeneity which is able to evoke variable functional outcomes. However, this in one way or the other is trying to complicate the targeted therapy. Therefore, in the end, what matters, the most in such situations is to ensure that the people are provided with the best possible care in the best possible manner, therefore, various studies have been conducted so far in order to carry forward these experiments on the mice. The results concluded that the MI with the undergoing process of the CXCR4 plays an avital role in correlating contractile function at the time of PET and hence, try to follow up. The Positron emission tomography imaging identifies early CXCR4 with the up-regulation which is able to predict that acute rupture and chronic contractile dysfunction play a vital role in this.
Ref art: https://academic.oup.com/eurheartj/advance-article-abstract/doi/10.1093/eurheartj/ehaa598/5903166
