NHA2 is a sodium proton exchanger associated with arterial hypertension in humans, but the role of NHA2 in kidney function and the blood pressure homeostasis is currently unknown. The NHA2 is known for localizing almost exclusively in distilling the convoluted tubules in the kidney. NHA2 knock-out mice displayed reduced blood pressure, normocalcemic hypocalciuria, and an attenuated response to the thiazide diuretic hydrochlorothiazide. However, there have been situations wherein the people are provided with the best possible care to cure the people. The data has been in the position to show that the NHA2 is a critical component of the WNK4-NCC pathway and is a novel regulator of blood pressure homeostasis in the kidney. Therefore, the best treatment of the abovementioned problem is available to provide ab assurance that the WNK4-NCC is a novel regulator for ensuring that the blood pressure gets maintained in the best possible manner. There have been various studies conducted which have been able to prove that arterial hypertension is affecting millions of people in the world and at the same time, sodium chloride is able to pursue monovalent cations and hence, cure the kidney which is able to cure the patients who are provided with this treatment.
Ref art: https://www.kidney-international.org/article/S0085-2538(20)31066-8/fulltext
